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Sortis (Atorvastatin)
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Sortis

Sortis is an HMG-CoA reductase inhibitor used to lower lipid levels and reduce the risk of cardiovascular disease including myocardial infarction and stroke. Sortis is indicated for the treatment of several types of dyslipidemias, including primary hyperlipidemia and mixed dyslipidemia in adults, hypertriglyceridemia, primary dysbetalipoproteinemia, homozygous familial hypercholesterolemia, and heterozygous familial hypercholesterolemia in adolescent patients with failed dietary modifications.

Other names for this medication:
Atacor, Atenfar, Ateroclar, Atorin, Atorvin, Atovarol, Atroact, Aztor, Biger, Cholvast, Finlipol, Holisten, Larus, Liparex, Lipibec, Lipidan, Lipitaksin, Lipium, Lipizem, Lipizim, Lipobi, Lipofin, Liponorm, Lipostatin, Lipostop, Liprimar, Locol, Lowlipen, Prevencor, Saphire, Sortis, Tiginor, Torid, Torivas, Totalip, Vasolip, Vastina, Xelitor, Zarator

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Also known as: Atorvastatin

Description

Generic Sortis is made by highly educated specialists to combat high cholesterol diseases (heart attack, stroke). Target of Generic Sortis is to control and decrease level of cholesterol. Generic Sortis acts as an anti-high cholesterol remedy. Generic Sortis operates by reducing decrease level of cholesterol. Sortis is also known as Atorvastatin, Atorbest, Agitor, Attor, Atorlip, Lipvas, Sortis, Torvast, Torvacard, Totalip, Tulip. Generic Sortis is HMG-CoA reductase inhibitor (statin). Generic name of Generic Sortis is Atorvastatin. Brand name of Generic Sortis is Sortis.

Dosage

Take Sortis exactly as prescribed by your doctor. Follow all directions on your prescription label and read all medication guides or instruction sheets. Your doctor may occasionally change your dose to make sure you get the best results. Do not use this medicine in larger or smaller amounts or for longer than recommended.

Sortis is usually taken once a day, with or without food. Take the medicine at the same time each day.

Do not break an Sortis tablet before taking it, unless your doctor has told you to.

You may need to stop using this medicine for a short time if you have:

uncontrolled seizures; an electrolyte imbalance (such as high or low potassium levels in your blood); severely low blood pressure; a severe infection or illness; or surgery or a medical emergency.

It may take up to 2 weeks before your cholesterol levels improve, and you may need frequent blood test. Even if you have no symptoms, tests can help your doctor determine if Sortis is effective.

Sortis is only part of a complete treatment program that may also include diet, exercise, and weight control. Follow your doctor's instructions very closely.

Store at room temperature away from moisture, heat, and light.

Detailed Sortis dosage information

Overdose

Seek emergency medical attention or call the Poison Help line at 1-800-222-1222.

Storage

Store at room temperature between 20 to 25 degrees C (68 to 77 degrees F) away from moisture and heat. Throw away any unused medicine after the expiration date. Keep out of the reach of children.

Side effects

The most common side effects associated with Sortis are:

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Side effect occurrence does not only depend on medication you are taking, but also on your overall health and other factors.

Contraindications

Avoid eating foods that are high in fat or cholesterol. Sortis will not be as effective in lowering your cholesterol if you do not follow a cholesterol-lowering diet plan.

Avoid drinking alcohol. It can raise triglyceride levels and may increase your risk of liver damage.

Grapefruit and grapefruit juice may interact with atorvastatin and lead to unwanted side effects. Avoid drinking more than 1 liter of grapefruit juice per day while taking this medicine.

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Spironolactone versus placebo, bisoprolol, and doxazosin to determine the optimal treatment for drug-resistant hypertension (PATHWAY-2): a randomised, double-blind, crossover trial. METABOLIC EFFECTS OF SYMPATHOMIMETIC AMINES PRODUCING CARDIAC LESIONS. Reciprocal projections were observed between the CoA and the MeA and between both nuclei and the basal or the lateral nuclei of the amygdala with the exception of the CoA which does not send inputs to the lateral nucleus. Although genetic testing prior to the initiation of irinotecan therapy is not currently recommended, assessment of UGT1A1 polymorphism is warranted when severe adverse events typical of systemic therapy manifest following DEBIRI-TACE. Mortality from ischemic cardiac disease in adults has been dramatically reduced by the development of novel therapies for inhibiting platelet function. Inhibition of the calpain system is most likely the mechanism by which corticosteroids induce this protective effect. Our results show that before nucleosome assembly is complete, H3K14 specificity is almost equal to that of H4K12 and DNA-histone interactions suppress the acetylation ability of Piccolo NuA4. The interaction of bone and glucose metabolism established by these studies will be important to a study of the two scientific fields in the future, particularly a clinical field.

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Extracellular vesicles (EVs) such as exosomes and microvesicles mediate intercellular communication and regulate a diverse range of crucial biological processes. Patients with delayed free flap surgery after lower leg trauma present a hypercoagulable state in their blood due to activation of the coagulation system and hypofibrinolysis. This chapter will describe treatment and host factors that influence immune function and how effective local control and systemic interventions of sarcoma therapy can cause inflammation and/or immune suppression but are currently the standard of care. Other studies showed that stimulus repetition suppresses the neural response. Recent studies have underscored the importance of inflammation in the development and progression of renal damage in renal artery stenosis. SURGICAL TREATMENT OF PORTAL HYPERTENSION DUE TO SCHISTOSOMIASIS. OCS impacts on health-related quality of life (HRQoL) have not been quantified. A significant difference was observed between Visit 1 and Visit 2, for endometrial volume, thickness, length and width in the pregnant group, and for endometrial volume, thickness and width in the non-pregnant group. Texting and walking: effect of environmental setting and task prioritization on dual-task interference in healthy young adults. These results support the notion that auditory discrimination is driven by the development of brain networks for predictive processing, and can be measured in infants during sleep.

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Observation for assessment of clinician performance: a narrative review. We have carried out a frying and cooking experiment, which has shown that the concentrations of PCBs and organochlorine pesticides (OCPs) increase in eel after frying. Enteral progesterone administration increased plasma levels of GLP-1, glucose-dependent insulinotropic polypeptide (GIP), and insulin, and improved oral glucose tolerance in an RU486-insensitve manner in mice: however, systemic progesterone exposure did not improve glucose homeostasis. Cardiac involvement can be detected in patients with hereditary ATTR amyloidosis with isolated neurological symptoms and without left ventricular wall thickening, suggesting that CMR could be useful in detecting preclinical cardiac amyloidosis. Charge transfer and coherent charge propagation in metal-insulator junctions. Extensive remodeling of a cyanobacterial photosynthetic apparatus in far-red light. Here we present the first study comparing all the paired appendages muscles of representatives of each major extant gnathostome group. The peptides and HLAs in the modules were characterized by similarity analysis.

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